Patient Case: Stasis Dermatitis
A 67-year-old woman presents with a six-month history of an itchy, red rash on both lower legs. She also reports leg swelling that worsens by the end of the day and improves with elevation. Over the past few weeks, the skin has become darker, scaly, and slightly thickened, with occasional weeping of fluid. She denies fever, trauma, or recent changes in skin care products.
Her medical history includes hypertension and varicose veins, but she has no known allergies or history of eczema. She does not smoke and has no history of diabetes.
On examination, there is bilateral leg edema, with erythematous, hyperpigmented patches on the medial lower legs. The skin appears dry, scaly, and slightly thickened, with mild oozing in some areas. There are no ulcers, but hemosiderin staining (brown discoloration) is visible. Peripheral pulses are intact, and there are no signs of infection. A clinical diagnosis of stasis dermatitis is made, likely due to chronic venous insufficiency.
She is advised to elevate her legs, use compression stockings, and apply moisturizers and mild topical corticosteroids to reduce inflammation. She is also referred for a vascular assessment to evaluate her venous circulation.
a) Cellulitis
b) Atopic dermatitis
c) Stasis dermatitis
d) Contact dermatitis
a) Chronic venous insufficiency
b) Arterial insufficiency
c) Fungal infection
d) Allergic reaction
a) Oral antibiotics and surgical debridement
b) Leg elevation, compression therapy, and topical corticosteroids
c) High-potency topical steroids and systemic immunosuppressants
d) Immediate venous surgery
a) Venous leg ulcers
b) Skin cancer
c) Rheumatoid arthritis
d) Atopic dermatitis
Answers
- (c) Stasis dermatitis
- The chronic bilateral lower leg rash, associated with swelling, hyperpigmentation, and scaling, is characteristic of stasis dermatitis. Cellulitis (a) presents with unilateral, acute, painful erythema with systemic symptoms. Atopic dermatitis (b) is more common in younger patients and affects flexural areas, while contact dermatitis (d) typically has a clear allergic or irritant trigger.
- (a) Chronic venous insufficiency
- Stasis dermatitis results from poor venous circulation, leading to fluid buildup, inflammation, and skin changes. Arterial insufficiency (b) causes pale, cold skin and ulcers on pressure points rather than an itchy, swollen rash. Fungal infections (c) typically cause localized, scaly lesions with central clearing, and allergic reactions (d) are acute with a known trigger.
- (b) Leg elevation, compression therapy, and topical corticosteroids
- The first-line treatment includes leg elevation and compression therapy to improve venous return, along with topical corticosteroids to reduce inflammation. Antibiotics and debridement (a) are needed only if infection or ulcers develop. High-potency steroids and immunosuppressants (c) are unnecessary, and venous surgery (d) is reserved for severe cases after conservative management fails.
- (a) Venous leg ulcers
- Untreated stasis dermatitis can progress to chronic venous ulcers, which are difficult to heal and prone to infection. Skin cancer (b), rheumatoid arthritis (c), and atopic dermatitis (d) are not direct complications of stasis dermatitis.
